Difference between revisions of "YOL032W"
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| − | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Systematic name''' || [http:// | + | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Systematic name''' || [http://www.yeastgenome.org/cgi-bin/locus.pl?dbid=S000005392 YOL032W] |
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|valign="top" nowrap bgcolor="{{SGDblue}}"| '''Gene name''' ||''OPI10 '' | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Gene name''' ||''OPI10 '' | ||
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|valign="top" nowrap bgcolor="{{SGDblue}}"| '''Coordinates''' | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Coordinates''' | ||
| − | |nowrap| Chr XV: | + | |nowrap| Chr XV:265429..266169 |
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| − | | | + | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Primary SGDID''' || S000005392 |
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| − | '''Description of | + | '''Description of YOL032W:''' Protein with a possible role in phospholipid biosynthesis, based on inositol-excreting phenotype of the null mutant and its suppression by exogenous choline<ref name='S000114750'>Hancock LC, et al. (2006) Genomic analysis of the Opi- phenotype. Genetics 173(2):621-34 |
{{SGDpaper|S000114750}} PMID 16582425</ref> | {{SGDpaper|S000114750}} PMID 16582425</ref> | ||
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==Community Commentary== | ==Community Commentary== | ||
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| + | <!-- PLEASE ADD Community Commentary ABOVE THIS MESSAGE. See below for an example of community annotation --> | ||
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| + | Specifically higher expression in carbon limited chemostat cultures versus carbon excess. | ||
| + | <ref>Boer VM, et al. (2003) The genome-wide transcriptional responses of Saccharomyces cerevisiae grown on glucose in aerobic chemostat cultures limited for carbon, nitrogen, phosphorus, or sulfur. | ||
| + | J Biol Chem 278(5):3265-74</ref> | ||
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==References== | ==References== | ||
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Latest revision as of 07:45, 23 January 2012
Share your knowledge...Edit this entry! <protect>
| Systematic name | YOL032W |
| Gene name | OPI10 |
| Aliases | |
| Feature type | ORF, Verified |
| Coordinates | Chr XV:265429..266169 |
| Primary SGDID | S000005392 |
Description of YOL032W: Protein with a possible role in phospholipid biosynthesis, based on inositol-excreting phenotype of the null mutant and its suppression by exogenous choline[1]
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Contents
Community Commentary
About Community Commentary. Please share your knowledge!
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References
See Help:References on how to add references
See Help:Categories on how to add the wiki page for this gene to a Category </protect>
