Difference between revisions of "YFL033C"

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'''Description of {{PAGENAME}}:''' Glucose-repressible protein kinase involved in signal transduction during cell proliferation in response to nutrients, specifically the establishment of stationary phase; originally identified as a regulator of IME2<ref name='S000080591'>Roosen J, et al. (2005) PKA and Sch9 control a molecular switch important for the proper adaptation to nutrient availability. Mol Microbiol 55(3):862-80 {{SGDpaper|S000080591}} PMID 15661010</ref><ref name='S000077209'>Cameroni E, et al. (2004) The novel yeast PAS kinase Rim 15 orchestrates G0-associated antioxidant defense mechanisms. Cell Cycle 3(4):462-8 {{SGDpaper|S000077209}} PMID 15300954</ref><ref name='S000047336'>Vidan S and Mitchell AP (1997) Stimulation of yeast meiotic gene expression by the glucose-repressible protein kinase Rim15p. Mol Cell Biol 17(5):2688-97
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'''Description of {{PAGENAME}}:''' Glucose-repressible protein kinase involved in signal transduction during cell proliferation in response to nutrients, specifically the establishment of stationary phase; identified as a regulator of IME2; substrate of Pho80p-Pho85p kinase<ref name='S000113685'>Wanke V, et al. (2005) Regulation of G0 entry by the Pho80-Pho85 cyclin-CDK complex. EMBO J 24(24):4271-8 {{SGDpaper|S000113685}} PMID 16308562</ref><ref name='S000080591'>Roosen J, et al. (2005) PKA and Sch9 control a molecular switch important for the proper adaptation to nutrient availability. Mol Microbiol 55(3):862-80 {{SGDpaper|S000080591}} PMID 15661010</ref><ref name='S000077209'>Cameroni E, et al. (2004) The novel yeast PAS kinase Rim 15 orchestrates G0-associated antioxidant defense mechanisms. Cell Cycle 3(4):462-8 {{SGDpaper|S000077209}} PMID 15300954</ref><ref name='S000047336'>Vidan S and Mitchell AP (1997) Stimulation of yeast meiotic gene expression by the glucose-repressible protein kinase Rim15p. Mol Cell Biol 17(5):2688-97
 
  {{SGDpaper|S000047336}} PMID 9111339</ref>
 
  {{SGDpaper|S000047336}} PMID 9111339</ref>
 
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Specifically higher expression in carbon limited chemostat cultures versus carbon excess.
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<ref>Boer VM, et al. (2003) The genome-wide transcriptional responses of Saccharomyces cerevisiae grown on glucose in aerobic chemostat cultures limited for carbon, nitrogen, phosphorus, or sulfur.
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J Biol Chem 278(5):3265-74</ref>
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Revision as of 13:02, 21 February 2007

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Systematic name YFL033C
Gene name RIM15
Aliases TAK1
Feature type ORF, Verified
Coordinates Chr VI:74425..69113


Description of YFL033C: Glucose-repressible protein kinase involved in signal transduction during cell proliferation in response to nutrients, specifically the establishment of stationary phase; identified as a regulator of IME2; substrate of Pho80p-Pho85p kinase[1][2][3][4]




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References

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  1. Wanke V, et al. (2005) Regulation of G0 entry by the Pho80-Pho85 cyclin-CDK complex. EMBO J 24(24):4271-8 SGD PMID 16308562
  2. Roosen J, et al. (2005) PKA and Sch9 control a molecular switch important for the proper adaptation to nutrient availability. Mol Microbiol 55(3):862-80 SGD PMID 15661010
  3. Cameroni E, et al. (2004) The novel yeast PAS kinase Rim 15 orchestrates G0-associated antioxidant defense mechanisms. Cell Cycle 3(4):462-8 SGD PMID 15300954
  4. Vidan S and Mitchell AP (1997) Stimulation of yeast meiotic gene expression by the glucose-repressible protein kinase Rim15p. Mol Cell Biol 17(5):2688-97 SGD PMID 9111339

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