Difference between revisions of "YLR320W"
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{|{{Prettytable}} align = 'right' width = '200px' | {|{{Prettytable}} align = 'right' width = '200px' | ||
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− | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Systematic name''' || [http://db.yeastgenome.org/cgi-bin/locus.pl? | + | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Systematic name''' || [http://db.yeastgenome.org/cgi-bin/locus.pl?dbid=S000004312 YLR320W] |
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|valign="top" nowrap bgcolor="{{SGDblue}}"| '''Gene name''' ||''MMS22 '' | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Gene name''' ||''MMS22 '' | ||
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|valign="top" nowrap bgcolor="{{SGDblue}}"| '''Coordinates''' | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Coordinates''' | ||
|nowrap| Chr XII:771940..776304 | |nowrap| Chr XII:771940..776304 | ||
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+ | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Primary SGDID''' || S000004312 | ||
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<br> | <br> | ||
− | '''Description of | + | '''Description of YLR320W:''' Protein involved in resistance to ionizing radiation; acts with Mms1p in a repair pathway that may be involved in resolving replication intermediates or preventing the damage caused by blocked replication forks<ref name='S000073328'>Araki Y, et al. (2003) Budding yeast mcm10/dna43 mutant requires a novel repair pathway for viability. Genes Cells 8(5):465-80 {{SGDpaper|S000073328}} PMID 12694535</ref><ref name='S000069458'>Bennett CB, et al. (2001) Genes required for ionizing radiation resistance in yeast. Nat Genet 29(4):426-34 |
{{SGDpaper|S000069458}} PMID 11726929</ref> | {{SGDpaper|S000069458}} PMID 11726929</ref> | ||
<br> | <br> | ||
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J Biol Chem 278(5):3265-74</ref> | J Biol Chem 278(5):3265-74</ref> | ||
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Revision as of 07:46, 27 February 2007
Share your knowledge...Edit this entry! <protect>
Systematic name | YLR320W |
Gene name | MMS22 |
Aliases | SLM2 |
Feature type | ORF, Verified |
Coordinates | Chr XII:771940..776304 |
Primary SGDID | S000004312 |
Description of YLR320W: Protein involved in resistance to ionizing radiation; acts with Mms1p in a repair pathway that may be involved in resolving replication intermediates or preventing the damage caused by blocked replication forks[1][2]
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