Difference between revisions of "YNL098C"
SGDwikiBot (talk | contribs) (Automated import of articles) |
SGDwikiBot (talk | contribs) (Automated import of articles) |
||
| Line 23: | Line 23: | ||
<br> | <br> | ||
<br> | <br> | ||
| + | === Alleles, Strains, and Phenotypes === | ||
| + | [[Category:Topic:Alleles, Strains, and Phenotypes]] | ||
| + | ==== Other Mutation ==== | ||
| + | [[Category:Topic:Alleles, Strains, and Phenotypes:Other Mutation]] | ||
| + | '''Strain Background''': W303-1B<br> | ||
| + | '''Together with''': YFH1<br> | ||
| + | '''Phenotype(s)''': Viable [[Category:Phenotype:Viable]] | ||
| + | '''Mutation type(s)''': transposon insertion | ||
| + | |||
| + | A C-terminal truncation of RAS2 increased mRNA levels of CIT2 by a factor of two. <ref name='S000075657'>Kucej M and Foury F (2003) Iron toxicity protection by truncated Ras2 GTPase in yeast strain lacking frataxin. Biochem Biophys Res Commun 310(3):986-91 {{SGDpaper|S000075657}} PMID 14550302</ref> <ref name = 'CAset3226-2004-03-26'>submitted by [http://db.yeastgenome.org/cgi-bin/colleague/colleagueSearch?id=3226 Martin Kucej] on 2004-03-26</ref> | ||
| + | |||
| + | {{ShortCenteredHR}} | ||
| + | === Interactions === | ||
| + | [[Category:Topic:Interactions]] | ||
| + | ==== Genetic ==== | ||
| + | [[Category:Topic:Interactions:Genetic]] | ||
| + | YNL098C is a suppressor of YFH1 [[Category:Genetic Interaction with YFH1]] | ||
| + | |||
| + | |||
| + | '''Strain Background''': W303-1B<br> | ||
| + | '''Together with''': YFH1<br> | ||
| + | '''Mutation type(s)''': partial deletion, deletion | ||
| + | |||
| + | A mutation in RAS2 supressed a growth defect of deltaYFH1 strain on toxic levels of iron in growth media. <ref name='S000075657'>Kucej M and Foury F (2003) Iron toxicity protection by truncated Ras2 GTPase in yeast strain lacking frataxin. Biochem Biophys Res Commun 310(3):986-91 {{SGDpaper|S000075657}} PMID 14550302</ref> <ref name = 'CAset3226-2004-03-26'>submitted by [http://db.yeastgenome.org/cgi-bin/colleague/colleagueSearch?id=3226 Martin Kucej] on 2004-03-26</ref> | ||
| + | |||
| + | {{ShortCenteredHR}} | ||
__TOC__ | __TOC__ | ||
==Community Commentary== | ==Community Commentary== | ||
{{CommentaryHelp}} | {{CommentaryHelp}} | ||
| + | === Alleles, Strains, and Phenotypes === | ||
| + | [[Category:Topic:Alleles, Strains, and Phenotypes]] | ||
| + | ==== Other Mutation ==== | ||
| + | [[Category:Topic:Alleles, Strains, and Phenotypes:Other Mutation]] | ||
| + | |||
| + | '''Strain Background''': W303-1B<br> | ||
| + | '''Together with''': YFH1<br> | ||
| + | '''Phenotype(s)''': Viable [[Category:Phenotype:Viable]] | ||
| + | '''Mutation type(s)''': transposon insertion | ||
| + | |||
| + | A C-terminal truncation of RAS2 increased mRNA levels of CIT2 by a factor of two. <ref name='S000075657'>Kucej M and Foury F (2003) Iron toxicity protection by truncated Ras2 GTPase in yeast strain lacking frataxin. Biochem Biophys Res Commun 310(3):986-91 {{SGDpaper|S000075657}} PMID 14550302</ref> <ref name = 'CAset3226-2004-03-26'>submitted by [http://db.yeastgenome.org/cgi-bin/colleague/colleagueSearch?id=3226 Martin Kucej] on 2004-03-26</ref> | ||
| + | |||
| + | {{ShortCenteredHR}} | ||
| + | === Interactions === | ||
| + | [[Category:Topic:Interactions]] | ||
| + | ==== Genetic ==== | ||
| + | [[Category:Topic:Interactions:Genetic]] | ||
| + | YNL098C is a suppressor of YFH1 [[Category:Genetic Interaction with YFH1]] | ||
| + | |||
| + | |||
| + | '''Strain Background''': W303-1B<br> | ||
| + | '''Together with''': YFH1<br> | ||
| + | '''Mutation type(s)''': partial deletion, deletion | ||
| + | |||
| + | A mutation in RAS2 supressed a growth defect of deltaYFH1 strain on toxic levels of iron in growth media. <ref name='S000075657'>Kucej M and Foury F (2003) Iron toxicity protection by truncated Ras2 GTPase in yeast strain lacking frataxin. Biochem Biophys Res Commun 310(3):986-91 {{SGDpaper|S000075657}} PMID 14550302</ref> <ref name = 'CAset3226-2004-03-26'>submitted by [http://db.yeastgenome.org/cgi-bin/colleague/colleagueSearch?id=3226 Martin Kucej] on 2004-03-26</ref> | ||
| + | {{ShortCenteredHR}} | ||
==References== | ==References== | ||
<!-- REFERENCES ARE AUTOMATICALLY GENERATED. PLEASE DON'T EDIT THIS SECTION--> | <!-- REFERENCES ARE AUTOMATICALLY GENERATED. PLEASE DON'T EDIT THIS SECTION--> | ||
{{RefHelp}} | {{RefHelp}} | ||
Revision as of 17:05, 23 January 2007
Share your knowledge...Edit this entry!
| Systematic name | YNL098C | |
| Gene name | RAS2 | |
| Aliases | CTN5, CYR3, GLC5, TSL7 | |
| Feature type | ORF, Verified | |
| Coordinates | Chr XIV:440572..439604 | |
| Don't edit this box! It's automatically regenerated, and edits will be lost when the update script runs. | ||
Description of YNL098C: GTP-binding protein that regulates the nitrogen starvation response, sporulation, and filamentous growth; farnesylation and palmitoylation required for activity and localization to plasma membrane; homolog of mammalian Ras proto-oncogenes[1][2][3]
Alleles, Strains, and Phenotypes
Other Mutation
Strain Background: W303-1B
Together with: YFH1
Phenotype(s): Viable
Mutation type(s): transposon insertion
A C-terminal truncation of RAS2 increased mRNA levels of CIT2 by a factor of two. [4] [5]
Interactions
Genetic
YNL098C is a suppressor of YFH1
Strain Background: W303-1B
Together with: YFH1
Mutation type(s): partial deletion, deletion
A mutation in RAS2 supressed a growth defect of deltaYFH1 strain on toxic levels of iron in growth media. [4] [5]
Contents
Community Commentary
About Community Commentary. Please share your knowledge!
Alleles, Strains, and Phenotypes
Other Mutation
Strain Background: W303-1B
Together with: YFH1
Phenotype(s): Viable
Mutation type(s): transposon insertion
A C-terminal truncation of RAS2 increased mRNA levels of CIT2 by a factor of two. [4] [5]
Interactions
Genetic
YNL098C is a suppressor of YFH1
Strain Background: W303-1B
Together with: YFH1
Mutation type(s): partial deletion, deletion
A mutation in RAS2 supressed a growth defect of deltaYFH1 strain on toxic levels of iron in growth media. [4] [5]
References
See Help:References on how to add references
- ↑ Bhattacharya S, et al. (1995) Ras membrane targeting is essential for glucose signaling but not for viability in yeast. Proc Natl Acad Sci U S A 92(7):2984-8 SGD PMID 7708760
- ↑ Kataoka T, et al. (1984) Genetic analysis of yeast RAS1 and RAS2 genes. Cell 37(2):437-45 SGD PMID 6327067
- ↑ Gimeno CJ, et al. (1992) Unipolar cell divisions in the yeast S. cerevisiae lead to filamentous growth: regulation by starvation and RAS. Cell 68(6):1077-90 SGD PMID 1547504
- ↑ 4.0 4.1 4.2 4.3 Kucej M and Foury F (2003) Iron toxicity protection by truncated Ras2 GTPase in yeast strain lacking frataxin. Biochem Biophys Res Commun 310(3):986-91 SGD PMID 14550302
- ↑ 5.0 5.1 5.2 5.3 submitted by Martin Kucej on 2004-03-26
See Help:Categories on how to add the wiki page for this gene to a Category
