YDR389W
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| Systematic name | YDR389W | |
| Gene name | SAC7 | |
| Aliases | ||
| Feature type | ORF, Verified | |
| Coordinates | Chr IV:1252529..1254493 | |
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Description of YDR389W: GTPase activating protein (GAP) for Rho1p, involved in signaling to the actin cytoskeleton, null mutations suppress tor2 mutations and temperature sensitive mutations in actin; potential Cdc28p substrate[1][2][3][4]
Protein Details
Protein Modification
Modification(s): Phosphorylation
Identified as an efficient substrate of Clb2-Cdk1-as1 in a screen of a proteomic GST-fusion library. [1] [5]
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Protein Details
Protein Modification
Modification(s): Phosphorylation
Identified as an efficient substrate of Clb2-Cdk1-as1 in a screen of a proteomic GST-fusion library. [1] [5]
References
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- ↑ 1.0 1.1 1.2 Ubersax JA, et al. (2003) Targets of the cyclin-dependent kinase Cdk1. Nature 425(6960):859-64 SGD PMID 14574415
- ↑ Schmidt A, et al. (2002) The RHO1-GAPs SAC7, BEM2 and BAG7 control distinct RHO1 functions in Saccharomyces cerevisiae. Mol Microbiol 45(5):1433-41 SGD PMID 12207708
- ↑ Dunn TM and Shortle D (1990) Null alleles of SAC7 suppress temperature-sensitive actin mutations in Saccharomyces cerevisiae. Mol Cell Biol 10(5):2308-14 SGD PMID 2183030
- ↑ Schmidt A, et al. (1997) The yeast phosphatidylinositol kinase homolog TOR2 activates RHO1 and RHO2 via the exchange factor ROM2. Cell 88(4):531-42 SGD PMID 9038344
- ↑ 5.0 5.1 submitted by Jeff Ubersax on 2004-01-21
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