YLR376C
Revision as of 07:21, 26 February 2009 by SGDwikiBot (talk | contribs) (Automated import of articles)
Share your knowledge...Edit this entry! <protect>
Systematic name | YLR376C |
Gene name | PSY3 |
Aliases | |
Feature type | ORF, Verified |
Coordinates | Chr XII:873553..872825 |
Primary SGDID | S000004368 |
Description of YLR376C: Protein involved in a Rad51p-, Rad54p-dependent pathway for homologous recombination repair; deletion results in a mutator phenotype; deletion increases sensitivity to anticancer drugs oxaliplatin and cisplatin but not mitomycin C[1][2][3]
</protect>
Contents
Community Commentary
About Community Commentary. Please share your knowledge!
<protect>
References
See Help:References on how to add references
- ↑ Mankouri HW, et al. (2007) Shu proteins promote the formation of homologous recombination intermediates that are processed by sgs1-rmi1-top3. Mol Biol Cell 18(10):4062-73 SGD PMID 17671161
- ↑ Wu HI, et al. (2004) Genome-wide identification of genes conferring resistance to the anticancer agents cisplatin, oxaliplatin, and mitomycin C. Cancer Res 64(11):3940-8 SGD PMID 15173006
- ↑ Huang ME, et al. (2003) A genomewide screen in Saccharomyces cerevisiae for genes that suppress the accumulation of mutations. Proc Natl Acad Sci U S A 100(20):11529-34 SGD PMID 12972632
See Help:Categories on how to add the wiki page for this gene to a Category </protect>