Difference between revisions of "YPR005C"
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| + | Specifically higher expression in carbon limited chemostat cultures versus carbon excess. | ||
| + | <ref>Boer VM, et al. (2003) The genome-wide transcriptional responses of Saccharomyces cerevisiae grown on glucose in aerobic chemostat cultures limited for carbon, nitrogen, phosphorus, or sulfur. | ||
| + | J Biol Chem 278(5):3265-74</ref> | ||
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Revision as of 12:02, 21 February 2007
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| Systematic name | YPR005C |
| Gene name | HAL1 |
| Aliases | |
| Feature type | ORF, Verified |
| Coordinates | Chr XVI:566668..565784 |
Description of YPR005C: Cytoplasmic protein involved in halotolerance; decreases intracellular Na+ (via Ena1p) and increases intracellular K+ by decreasing efflux; expression repressed by Ssn6p-Tup1p and Sko1p and induced by NaCl, KCl, and sorbitol through Gcn4p[1][2][3][4]
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References
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- ↑ Pascual-Ahuir A, et al. (2001) The Sko1p repressor and Gcn4p activator antagonistically modulate stress-regulated transcription in Saccharomyces cerevisiae. Mol Cell Biol 21(1):16-25 SGD PMID 11113177
- ↑ Marquez JA, et al. (1998) The Ssn6-Tup1 repressor complex of Saccharomyces cerevisiae is involved in the osmotic induction of HOG-dependent and -independent genes. EMBO J 17(9):2543-53 SGD PMID 9564037
- ↑ Rios G, et al. (1997) Mechanisms of salt tolerance conferred by overexpression of the HAL1 gene in Saccharomyces cerevisiae. Yeast 13(6):515-28 SGD PMID 9178503
- ↑ Gaxiola R, et al. (1992) A novel and conserved salt-induced protein is an important determinant of salt tolerance in yeast. EMBO J 11(9):3157-64 SGD PMID 1505513
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