Difference between revisions of "YER028C"
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|valign="top" nowrap bgcolor="{{SGDblue}}"| '''Coordinates''' | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Coordinates''' | ||
| − | |nowrap| Chr V: | + | |nowrap| Chr V:211876..210692 |
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|valign="top" nowrap bgcolor="{{SGDblue}}"| '''Primary SGDID''' || S000000830 | |valign="top" nowrap bgcolor="{{SGDblue}}"| '''Primary SGDID''' || S000000830 | ||
Revision as of 13:05, 3 February 2011
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| Systematic name | YER028C |
| Gene name | MIG3 |
| Aliases | |
| Feature type | ORF, Verified |
| Coordinates | Chr V:211876..210692 |
| Primary SGDID | S000000830 |
Description of YER028C: Probable transcriptional repressor involved in response to toxic agents such as hydroxyurea that inhibit ribonucleotide reductase; phosphorylation by Snf1p or the Mec1p pathway inactivates Mig3p, allowing induction of damage response genes[1]
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Contents
Community Commentary
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DNA and RNA Details
Other DNA and RNA Details
Other Topic: expression
Specifically lower expression in carbon limited chemostat cultures versus carbon excess. [2] [3]
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References
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- ↑ Dubacq C, et al. (2004) The protein kinase Snf1 is required for tolerance to the ribonucleotide reductase inhibitor hydroxyurea. Mol Cell Biol 24(6):2560-72 SGD PMID 14993292
- ↑ Boer VM, et al. (2003) The genome-wide transcriptional responses of Saccharomyces cerevisiae grown on glucose in aerobic chemostat cultures limited for carbon, nitrogen, phosphorus, or sulfur. J Biol Chem 278(5):3265-74 SGD PMID 12414795
- ↑ submitted by Viktor Boer on 2003-07-25
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